What is attention? How does it go wrong? Do attention deficits arise from genes or from the environment? Can we cure it with drugs or training? Are there disorders of attention other than deficit disorders? The past decade has seen a burgeoning of research on the subject of attention. This research has been facilitated by advances on several fronts: New methods are now available for viewing brain activity in real time, there is expanding information on the complexities of the biochemistry of neural activity, individual genes can be isolated and their functions identified, analysis of the component processes included under the broad umbrella of "attention" has become increasingly sophisticated, and ingenious methods have been devised for measuring typical and atypical development of these processes, from infancy into childhood, and then into adulthood. In this book, Kim Cornish and John Wilding are concerned with attention and its development, both typical and atypical, particularly in disorders with a known genetic etiology or assumed genetic linkage. Tremendous advances across seemingly diverse disciplines - molecular genetics, pediatric neurology, child psychiatry, developmental cognitive neuroscience, and education - have culminated in a wealth of new methods for elucidating disorders at multiple levels, possibly paving the way for new treatment options. Cornish and Wilding use three specific-yet-interlinking levels of analysis: genetic blueprint (genotype), the developing brain, and the behavioral-cognitive outcomes (phenotype), as the basis for charting the attention profiles of six well-documented neurodevelopmental disorders: ADHD, autism, fragile X syndrome, Down syndrome, Williams syndrome, and 22q11 deletion syndrome. Their overarching aim in this book is to provide the most authoritative and extensive account to date of disorder-specific attention profiles and their development from infancy through adolescence.
... 165 Thomson , C. , 52 , 59 Thurman , S. K. , 230 Thyer , B. A. , 308 Timberlake , W. , 165 Webster - Stratton , 237 , 251 , 253 , 366 Author Index.
Haberstick, B.C., Lessem, J. M., Hopfer, C. J., Smolen, A., Ehringer, M.A., Timberlake, D., et al. (2005). Monoamine oxidase A (MAOA) and antisocial ...
Some, like the “behavior systems” approach of Timberlake(1994)assume thatbehavior can be explained by a system of interactingmodules thatareeither built ...
However, there is clear evidence that this constant ratio does not always produce reinforcement (Timberlake & Allison, 1974). Second and, as we shall see ...
... 30, 32 Thomae, H., 40 Thompson, L., 23-24 Timberlake, E. M., 16 Tobin, S. S., ... E, 33 Wolfe, S. M., 81 Wolinsky, M. A., 85 Zarit, J., 11, 30, 31, 32, ...
La Crisi Mondiale e Saggi Critici di Marxiano e Socialismo. Bologna, N. Zanichelli. ... TIMBERLAKE (P. H.): 1912. Experimental Parasitism, a Study of the ...
... 143 Tharp, R. G., 80 Thompson, R. H., 250 Timberlake, W., 308,309 Tingey, ... B. W., 70 Ries, B.J., 268 Robins, E.,298 Robinson, S. L., 91,244 Roper, ...
... R.L., McGrath, Joseph E. McKeachie McPhail, Clark Miller, J.G. Mitchell, ... Jerry 469 Taylor 39 Timberlake, William 464 Tolman 72, 140, 142 Tucker, ...
... 247 Fromme, H., 523 Frost, P., 106 Frost, R., 161 Fryer, R., 291 Fuhrer, D., 4 Fukuyama, H., 408 Fulbright, R. K., 486 Fulero, S., 440 Fuligni, A. J., ...
... C. 638 Ernst, D. 704 Ernst, E. 278 Esch, T. 110 Eslinger, P.J. 448 Esposito-Smythers, ... E. 197 Frontera, W. R. 408 Frost, J. 332 Frost, R. 699 Frost, ...