An updated and expanded edition on the roles that brain function and genetics play in addiction. Over the past 10 years, neurobiologic and genetic research has provided an increased understanding of what causes drug addiction in the brain’s reward pathway. Knowing this leads to a better understanding of how it may be treated and even reversed in those who successfully overcome the disease. This is especially true with addiction’s possible precursors of mild to moderate substance use disorders. These latter disorders can usually be treated more easily by less intensive models of “treatment” that do not require actual brain chemistry re-regulation over time. In this new edition, there are updated scientific references to support addiction as a medical brain disease, using the prevailing neurobiology, genetics, and psychological scientific literature. We now have more psychosocial and medicinal methods for reversing abnormal brain chemistry during drug addiction. There are also more effective intervention, counseling, and motivating methods (SBIRT, motivational interviewing) for overcoming resistance to treatment and resistance to change than were able to be discussed when the first edition was published over a decade ago. Here, readers will find a fully-updated glossary of terms, additional abbreviations, and updated appendices. These will aid in clarifying the somewhat lengthy and science-based upgrades in our knowledge of neuroscience and genetics research that are so critical in understanding why addiction is such a serious and tough-to-treat disease. Utilizing the same easy-to-read language that was a hallmark of the earlier edition, Erickson keeps the science understandable yet comprehensive—appropriate for health professionals as well as lay readers who need and want this critical information.
... 165 Thomson , C. , 52 , 59 Thurman , S. K. , 230 Thyer , B. A. , 308 Timberlake , W. , 165 Webster - Stratton , 237 , 251 , 253 , 366 Author Index.
Haberstick, B.C., Lessem, J. M., Hopfer, C. J., Smolen, A., Ehringer, M.A., Timberlake, D., et al. (2005). Monoamine oxidase A (MAOA) and antisocial ...
Some, like the “behavior systems” approach of Timberlake(1994)assume thatbehavior can be explained by a system of interactingmodules thatareeither built ...
However, there is clear evidence that this constant ratio does not always produce reinforcement (Timberlake & Allison, 1974). Second and, as we shall see ...
... 30, 32 Thomae, H., 40 Thompson, L., 23-24 Timberlake, E. M., 16 Tobin, S. S., ... E, 33 Wolfe, S. M., 81 Wolinsky, M. A., 85 Zarit, J., 11, 30, 31, 32, ...
La Crisi Mondiale e Saggi Critici di Marxiano e Socialismo. Bologna, N. Zanichelli. ... TIMBERLAKE (P. H.): 1912. Experimental Parasitism, a Study of the ...
... 143 Tharp, R. G., 80 Thompson, R. H., 250 Timberlake, W., 308,309 Tingey, ... B. W., 70 Ries, B.J., 268 Robins, E.,298 Robinson, S. L., 91,244 Roper, ...
... R.L., McGrath, Joseph E. McKeachie McPhail, Clark Miller, J.G. Mitchell, ... Jerry 469 Taylor 39 Timberlake, William 464 Tolman 72, 140, 142 Tucker, ...
... 247 Fromme, H., 523 Frost, P., 106 Frost, R., 161 Fryer, R., 291 Fuhrer, D., 4 Fukuyama, H., 408 Fulbright, R. K., 486 Fulero, S., 440 Fuligni, A. J., ...
... C. 638 Ernst, D. 704 Ernst, E. 278 Esch, T. 110 Eslinger, P.J. 448 Esposito-Smythers, ... E. 197 Frontera, W. R. 408 Frost, J. 332 Frost, R. 699 Frost, ...